https://repositorio.fleni.org.ar/xmlui/handle/123456789/385 2026-04-05T19:32:08Z https://repositorio.fleni.org.ar/xmlui/handle/123456789/1370 Long‑term clinical results of the Flow ventricular catheter for hydrocephalus: brief report. Galarza, Marcelo; Sosa, Fidel; Etus, Volkan; Argañaraz, Romina; Gazzeri, Roberto; Giménez, Ángel; Amigó, José María 2024-01-01T00:00:00Z https://repositorio.fleni.org.ar/xmlui/handle/123456789/1182 Spinal Cord UBOs in NF1 Pediatric Patients: Single Institution Experience Lombardi, Francina; Aguilar, Martín Santiago; Palomar, Nicolás; Carnevale, Martín Diego; Vigliano, Alejandra; Casola, A.; Schteinschnaider, Ángeles; Diez, Blanca Introduction: Neurofibromatosis type 1 (NF1) is a neurocutaneous disease that shows specific clinical characteristics and imaging. Being a predisposition syndrome to diferent tpes of tumors, neurofibromas and glioma of the optic pathway being the most common lesions. It is essential to know the typical, non-oncological lesions that can be present and diagnosed through neuroimaging, so that they are not missdiagnosed and treated incorrectly. Within these lesion we foun UBOs (Unidentified bright objects), located on white matter areas in the parenchyma, but little taken into account at spinal cord level. Aims: To describe the prevalence of UBOs located in the spinal cord, in patients with NF1 during childhood, based on data obtained at our institution. Materials and methods: Retrospective, observational and descriptive study of patients between 2 and 21 years old with NF1 diagnosis, evaluated during 2009 to 2023 time period, who had whole brain and spinal cord MRI (n=32). The presence of UBOs was evaluated. We refer to spinal UBO as single or multiple lesions located in the cord, hyperintense on T2/STIR sequences and isointense on T1, without gadolinium enhancement or mass effect. Results: Male patient 58.3%, with an average age of 13.1 years. Twenty seven patients (79%) had brain UBOs and 9 (26.4%) Spinal cord UBOs. Of the latter, 44.4% presented a single spinal cord lesion, the rest had more than one. The most comon location was the subaxial cervical (C3-T1) area, followed by the cervical superior (C1-C2) area, dorsal with 2 lesions and conus medullaris in only one case. In all of them, hypersignal was found in T2 sequence and isointense signal in T1; no case showed enhancement with intravenous contrast or mass effect. There was coexistence of brain UBOs in all our patients. None of the patients presented clinical manifestations associated with these lesions. Conclusion: There is little literature regarding the existence of spinal UBOs. It is important to recognize the existence and diagnostic imaging characteristics of these in order to avoid diagnostic and therapeutic errors regarding the presence of spinal cord lesion in NF1 patients. 2024-01-01T00:00:00Z https://repositorio.fleni.org.ar/xmlui/handle/123456789/943 FLAMES (FLAIR Hyperintense Lesions in Anti-Myelin Oligodendrocyte Glycoprotein associated Encephalitis With Seizures), a case report. González, María Gala; Dominguez, Romina Nélida; Yañez, Paulina; Massaro, Mario; Schteinschnaider, Ángeles 2022-10-03T00:00:00Z https://repositorio.fleni.org.ar/xmlui/handle/123456789/384 Pediatric posterior ischemic stroke due to compression of vertenral artery: bow hunter’s syndrome Biaggi, Leticia; Fulco, Vanesa; Bembenuto, L.; Routaboul, Carlos; Chaves, Hernán; Aversa, L.; Ameriso, Sebastián Francisco; Dossi, Daiana Elizabeth; Schteinschnaider, Ángeles Aims & Objectives: Acute traumatic stroke of the cerebellum is rarely seen in children. It may occur after minor trauma or by rotational movements of the head. Causes may include compression and/or injury of the posterior vertebral artery secondary to bone defect. One particular form, “Bow Hunter’s stroke”, originally described by Sorensen in 1978, results from vertebral-basilar insufficiency induced by head movements causing intermittent vertebral artery compression at the atlanto-axial junction. Acute ischemic lesions in this vascular territory should be suspected, and investigated using imaging studies such as CT and brain MRI. Methods: We present a case of a 7-year-old male with unremarkable medical history, with subacute left hemiparesis. MRI shows acute ischemic stroke secondary to dissection of the posterior vertebral artery, then confirmed by conventional angiography. CT reconstruction showed presence of a bone spur compressing the spinal canal at the level of the Occipito-axial junction. Although anticoagulation was prescribed, a second event occurred 20 days later, when he experienced vomiting and loos of consciousness. After surgical resection of the bone defect, patient post-operative recovery was uneventful and no further ischemic events were observed. Results: Surgical resolution Conclusions: Underlying causes or structural anomalies predisposing to stroke should be carefully evaluated and ruled out, since most are treatable and recurrences can be prevented, as in this case. 2021-03-01T00:00:00Z